The new theory of cardiovascular disease is based on the fact, that the arterial and venous pools may periodically be linked by large anastomoses (natural shunts) [2-6]. In normal conditions, arteriovenous anastomoses (AVA) are closed, but during periods of increased physical and psychological stress due to increased blood pressure, they can open. The most common pathological role extended AVA manifests in the liver area. More often, the pathological role of enlarged AVA is manifested in the region of the liver, for example, between the superior mesenteric artery and portal vein [7]. This leads to a reset of the arterial blood directly into veins. Because of this, some groups of working cells are left without sufficient food and O2. Under certain conditions, the AVA may be open too long. As a result, blood high pressure penetrates into the venous bed and gradually fills it. Usually first affecting the liver and vessels.